![Antioxidants | Free Full-Text | Endothelium as a Source and Target of H2S to Improve Its Trophism and Function Antioxidants | Free Full-Text | Endothelium as a Source and Target of H2S to Improve Its Trophism and Function](https://www.mdpi.com/antioxidants/antioxidants-10-00486/article_deploy/html/images/antioxidants-10-00486-g001.png)
Antioxidants | Free Full-Text | Endothelium as a Source and Target of H2S to Improve Its Trophism and Function
![Antioxidants | Free Full-Text | Endothelium as a Source and Target of H2S to Improve Its Trophism and Function Antioxidants | Free Full-Text | Endothelium as a Source and Target of H2S to Improve Its Trophism and Function](https://www.mdpi.com/antioxidants/antioxidants-10-00486/article_deploy/html/images/antioxidants-10-00486-g002.png)
Antioxidants | Free Full-Text | Endothelium as a Source and Target of H2S to Improve Its Trophism and Function
![Inhibition of cystathionine β-synthase promotes apoptosis and reduces cell proliferation in chronic myeloid leukemia | Signal Transduction and Targeted Therapy Inhibition of cystathionine β-synthase promotes apoptosis and reduces cell proliferation in chronic myeloid leukemia | Signal Transduction and Targeted Therapy](https://media.springernature.com/lw685/springer-static/image/art%3A10.1038%2Fs41392-020-00410-5/MediaObjects/41392_2020_410_Fig4_HTML.png)
Inhibition of cystathionine β-synthase promotes apoptosis and reduces cell proliferation in chronic myeloid leukemia | Signal Transduction and Targeted Therapy
![H2S Protects Against Pressure Overload–Induced Heart Failure via Upregulation of Endothelial Nitric Oxide Synthase | Circulation H2S Protects Against Pressure Overload–Induced Heart Failure via Upregulation of Endothelial Nitric Oxide Synthase | Circulation](https://www.ahajournals.org/cms/asset/a279c098-63d1-4c4c-b1b7-6168995b5666/1116fig01.jpeg)
H2S Protects Against Pressure Overload–Induced Heart Failure via Upregulation of Endothelial Nitric Oxide Synthase | Circulation
![H2S Protects Against Pressure Overload–Induced Heart Failure via Upregulation of Endothelial Nitric Oxide Synthase | Circulation H2S Protects Against Pressure Overload–Induced Heart Failure via Upregulation of Endothelial Nitric Oxide Synthase | Circulation](https://www.ahajournals.org/cms/asset/b93b4b2d-0f3b-44fa-8cc8-3aaba92b8321/1116fig03.jpg)
H2S Protects Against Pressure Overload–Induced Heart Failure via Upregulation of Endothelial Nitric Oxide Synthase | Circulation
![Inhibition of cystathionine β-synthase promotes apoptosis and reduces cell proliferation in chronic myeloid leukemia | Signal Transduction and Targeted Therapy Inhibition of cystathionine β-synthase promotes apoptosis and reduces cell proliferation in chronic myeloid leukemia | Signal Transduction and Targeted Therapy](https://media.springernature.com/lw685/springer-static/image/art%3A10.1038%2Fs41392-020-00410-5/MediaObjects/41392_2020_410_Fig5_HTML.png)
Inhibition of cystathionine β-synthase promotes apoptosis and reduces cell proliferation in chronic myeloid leukemia | Signal Transduction and Targeted Therapy
![H2S Protects Against Pressure Overload–Induced Heart Failure via Upregulation of Endothelial Nitric Oxide Synthase | Circulation H2S Protects Against Pressure Overload–Induced Heart Failure via Upregulation of Endothelial Nitric Oxide Synthase | Circulation](https://www.ahajournals.org/cms/asset/661a97a4-4610-4298-bb7a-5b14cad5fda6/1116fig06.jpg)